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. Author manuscript; available in PMC: 2016 Feb 2.
Published in final edited form as: Nanotoxicology. 2015 Sep 4;9(8):941–951. doi: 10.3109/17435390.2014.984251

Figure 2.

Figure 2

Mechanical initiators arteriolar responsiveness. Myogenic index calculations for (A) coronary (N = 8 (sham) and 13 (prenatally exposed) rats; n = 11 (sham) and 15 (prenatally exposed) vessels) and (B) uterine (N = 7 (sham) and 10 (prenatally exposed) rats; n = 8 (sham) and 11 (prenatally exposed) vessels) arterioles. Values are means ± SE. *p ≤ 0.05 two-way ANOVA; arteriolar dilation as a function of shear stress of the (C) coronary (N = 7 (sham) and 8 (exposed in utero) rats; n = 11 (sham) and 15 (exposed in utero) vessels) and (D) uterine (N = 7 (sham) and 11 (exposed in utero) rats; n = 11 (sham) and 11 (exposed in utero) vessels) arterioles. *p ≤ 0.05 sham regression line vs. gestational exposure regression line for both the coronary and uterine arterioles.