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. 2016 Feb 4;23:24. doi: 10.1186/s12929-016-0242-7

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© Chen et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — Urokinase-type plasminogen activator (uPA)-deficient mice failed to generate a type 2 immune response in a focal segmental glomerulosclerosis (FSGS) mouse model. Plasma samples in the wild-type (WT) and uPA^−/− groups were collected at indicated time points to determine levels of immunoglobulin G1 (IgG1), IgG2a, and IgG3. Data are presented as the ratio of IgG1 over the combination of IgG2a and IgG3. ^# p < 0.05 vs. week 0 (W0); *p < 0.05 vs. the WT