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(a) K‐ras wild‐type (WT) or mutant 3′‐UTR was subcloned into a luciferase reporter vector and cotransfected with miR‐1 or scrambled control into C666‐1 cells. (b) miR‐1 significantly inhibited the luciferase activity of the K‐ras WT 3′‐UTR but not that of the mutant in C666‐1 cells. (c) Overexpression of miR‐1 significantly downregulated K‐ras protein levels. *represent significance.
