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. 2015 Mar 19;119(10):1202–1209. doi: 10.1152/japplphysiol.01043.2014

Fig. 2.

Fig. 2.

A: arterial Pco2 (PaCO2), mean arterial blood pressure (MABP), and cortical laser-Doppler flow (LDF) are sorted by range of arterial O2 saturation and are plotted as means and 95% confidence intervals for groups treated with vehicle (n = 20), the EET synthesis inhibitor N-methylsulfonyl-6-(2-propargyloxyphenyl)hexanamide (MS-PPOH; n = 13), or the EET antagonist 14,15-epoxyeicosa-5(Z)-enoic acid (14,15-EEZE; n = 13). B: hypoxic responsivity, derived from the regression slope of the relative change of LDF vs. the reciprocal of arterial O2 content for each rat, is plotted for groups treated with vehicle, MS-PPOH, and 14,15-EEZE. *P < 0.05 from vehicle group.