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. 2016 Feb 9;35:30. doi: 10.1186/s13046-015-0262-2

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© Zhou et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Linc00152 highly correlated with EGFR and constitutively activated PI3K/AKT signaling. a Different expression of EGFR mRNA in 72 pairs tissues from gastric cancer patients. b The protein expression level of EGFR in patients with gastric cancer. c, d Patients was divided into Linc00152^high and Linc00152^low groups based on the median of Linc00152 expression. The mRNA (panel c) and protein expression (panel d) of EGFR were compared in the two groups. e Pearson analysis was performed in calculating the correlation of Linc00152 and EGFR with log-transformed data. f The activation of PI3K/AKT signaling was measured by detecting the p-EGFR, p-PI3K and p-AKT in both cells line and tumors from Xenograft model. Data was presented with mean ± SEM