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. 2016 Feb 16;16:4. doi: 10.1186/s40644-016-0062-8

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© Boussouar et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Poorly differentiated HCC in a patient with alcoholic cirrhosis, showing restricted ADC on DWI-MRI and increased uptake on the FDG-PET/CT. Transverse dynamic contrast-enhanced initial post contrast MR image shows a 30 mm large nodular enhancing lesion in the right liver on 3D GE arterial phase imaging (a, arrow) with wash-out on portal-venous phase (b, arrow). The lesion is mildly hyper intense on transverse DW image (b = 800 s/mm²; c, arrow) and has a low ADC (1.06 × 10⁻³ mm²/s) on the ADC map (d, arrow). FDG-PET/CT shows an increased tumour uptake with a SUV_T = 4.1 and SUV_T/L = 1.24 (e, arrow). The explanted liver showed an Edmondson-Steiner grade 4 HCC (f, arrow)