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. 2016 Feb 16;16:4. doi: 10.1186/s40644-016-0062-8

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© Boussouar et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Moderately differentiated (grade 2) HCC in a patient with hepatitis B-induced cirrhosis, showing restricted ADC on DWI-MRI and no significant uptake on FDG-PET/CT. MR imaging showed a left lobe HCC with high T1 (a, arrow), high T2 (b, arrow) signal intensity, with arterial enhancement (c, arrow) followed by washout (d, arrow). The HCC showed high signal intensity on DWI image (b = 800 s/mm²; e, arrow) consistent with low ADC (1.26 × 10⁻³ mm²/s) on the ADC map (f, arrow). The lesion showed no significant FDG uptake on FDG-PET/CT (g)