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Genetic mechanism for reported recurrent fusions ESR1-CCDC170 and MAGI3-AKT3 in breast cancer subtypes, ER+ and TNBC, respectively. (A) Tandem duplication as potential mechanism of generation of ESR1-CCDC170 gene fusion in breast cancers.95 (B) MAGI3-AKT3 fusion as a result of balanced translocation from intron 9 in MAGI3 to intron 1 in AKT3.96
