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. Author manuscript; available in PMC: 2017 Mar 15.
Published in final edited form as: Int J Cardiol. 2016 Jan 7;207:326–334. doi: 10.1016/j.ijcard.2016.01.016

Figure 5.

Figure 5

Computer simulated decrease of the peak sodium channel current (INa) during the train of pulses (5 and 200 ms duration) delivered from −80 mV to −30 mV in the presence of 10 g/L Wenxin Keli using diastolic interval of 150 ms. Peaks of INa simulated in the presence of Wenxin Keli were normalized to peaks of INa simulated during the same trains in control conditions. Data were simulated using an atrial sodium channel model (top panels; n=4) and using a ventricular sodium channel model (bottom panels; n=5) using pulse durations of 5 ms (A and C) and 200 ms (B and D). Compare with Figure 1.