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. 2015 Nov 11;115(1):345–354. doi: 10.1152/jn.00775.2015

Fig. 10.

Fig. 10.

IH facilitates CaV3.2 protein trafficking. A: representative Western blot (top) and densitometric analysis (bottom) of plasma membrane fractions of CaV3.2 and pan-cadherin (marker of membrane fraction) proteins of normoxia- and IH60-treated CaV3.2-HEK cells in the presence and absence of 30 ng/ml brefeldin A (BFA), a blocker of protein trafficking. B: densitometric analysis of Western blots of plasma membrane fractions of CaV3.2 expressed as a percentage of total cell CaV3.2 protein of normoxia- and IH60-treated CaV3.2-HEK cells in the presence and absence of 30 ng/ml BFA. C: average (means ± SE) normalized peak current density obtained with a step to −30 mV of CaV3.2-HEK cells treated with normoxia (n = 17 cells), IH60 (n = 18 cells), and IH60 + 30 nM BFA (n = 19 cells). **P < 0.01; n.s., not significant (P > 0.05).