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. Author manuscript; available in PMC: 2017 Feb 17.
Published in final edited form as: Neuron. 2016 Feb 4;89(4):741–755. doi: 10.1016/j.neuron.2016.01.020

Figure 5. Sema-2b genetically interacts with TORC2 complex and Trc kinase.

Figure 5

(A–J) Dendritic patterns of torΔP/+ (A), sema-2bC4/torΔP (B), rictorΔ2/+ (C), sema-2bC4/rictorΔ2 (D), sin1e03756/+ (E), sema-2bC4/sin1e03756 (F), trc1/+ (G), sema-2bC4/trc1(H), fry1/+ (I), and sema-2bC4/fry1 (J) class IV da neurons. Dendritic crossings are indicated by blue arrowheads. Scale bars represent 30 μm.

(K) Number of total crossing points normalized to total dendritic length in sema-2bC4/+ (n=5), torΔP/+ (n=4), sema-2bC4/torΔP (n=5), rictorΔ2/+ (n=5), sema-2bC4/rictorΔ2 (n=5), sin1e03756/+ (n=5), sema-2bC4/sin1e03756 (n=7), trc1/+ (n=4), sema-2bC4/trc1 (n=4), fry1/+ (n=5), and sema-2bC4/fry1 (n=5) neurons. White bars represent the quantifications of contacting crossings and red bars represent the quantification of contacting crossings. Data are plotted as average ± SEM. **p < 0.01, and ***p < 0.001 as assessed by one-way analysis of variance and Bonferroni test.