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. 2016 Feb 20;10:18. doi: 10.1186/s12918-016-0256-5

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© Chen and Li. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — The integrated genetic and epigenetic cellular network for investigating the cellular mechanisms of normal and tumor tissues in gastric cancer. The associations include the networks in tumor cells (dotted lines), normal cells (dashed lines), and both (solid lines). The bold lines indicate large genetic and epigenetic regulatory effects or genetic expression. Dysregulation of MIR7 contributes to initiation and progression of inflammation-induced gastric cancer, and PML, a potential drug target, overcomes platinum resistance in platinum-based chemotherapy in gastric cancer