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. 2016 Feb 20;10:18. doi: 10.1186/s12918-016-0256-5

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© Chen and Li. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — The integrated genetic and epigenetic cellular network for investigating the cellular mechanisms of normal and HIV-infected T cells at the intermediate HIV infection stage, i.e., the integration/replication stage. The associations include those in HIV-infected cells (dotted lines), normal cells (dashed lines), and both cell types (solid lines). The bold lines indicate large genetic and epigenetic regulatory effects or genetic expression. Dysregulation of MIR139-5p, and MIRLET7i contributes to dysfunction of the immune response and viral replication via DNA hypermethylation