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. 2016 Mar;89(3):323–334. doi: 10.1124/mol.115.101758

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Copyright © 2016 by The American Society for Pharmacology and Experimental Therapeutics

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Fig. 6. — Activation of endogenous βARs in A7r5 cells enhances exogenous hKv7.5, hKv7.4 and hKv7.4/7.5 to varying degrees. (A–C) Current-voltage curves of steady-state Kv currents recorded in A7r5 cells overexpressing hKv7.5 (A, n = 6), hKv7.4 (B, n = 6), or hKv7.4/7.5 (C, n = 5) before (control, solid circles) and after 5 minutes of treatment with 1 μM isoproterenol (open circles). The asterisks (*) indicate significant difference from control, P < 0.05, paired Student’s t test. (D–F) Conductance-voltage relationships of exogenous hKv7.5 (D), Kv7.4 (E), and Kv7.4/7.5 (F) channels normalized to maximal conductance (G_max) before (solid circles, n = 5-6) and after addition of 1 μM isoproterenol (open circles, n = 5–6) fitted to the Boltzmann equation (solid lines); conductances normalized to the maximal control conductance (G_{max_c}) are shown in the insets.