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. 2016 Jan 8;16(1):1–17. doi: 10.1007/s40268-015-0121-9

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© The Author(s) 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 International License (http://creativecommons.org/licenses/by-nc/4.0/), which permits any noncommercial use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 1 — Prominent examples of the broad array of therapeutic strategies that are showing promise for the instigation of normal expression and function of cystic fibrosis transmembrane regulator (CFTR), and their cellular targets. Ata ataluren, GP67a-pDNA lipid-enclosed DNA plasmid, Iva ivacaftor, Lum lumacaftor, ZFN zinc finger nuclease