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. Author manuscript; available in PMC: 2016 Feb 26.
Published in final edited form as: Curr Mol Med. 2014;14(6):703–711. doi: 10.2174/1566524014666140724095112

Fig.1. Ablation of Frs2α in hepatocytes does not cause apparent liver defects.

Fig.1

(A) In situ hybridization showing Frs2α expression (purple staining) in the liver of Frs2αFlox but not in Frs2αCN liver. Sense probe was used as a negative control. (B) Average body weight and the liver/body weight ratios of Frs2αFlox and Frs2αCN mice at the age of 2 months. (C) Gross morphology of Frs2αFlox and Frs2αCN liver of 2-month-old mice. (D) H&E staining showing no obvious histological difference between Frs2αFlox and Frs2αCN livers. (E) Ki67 staining showing no proliferative differences between two groups. Flox, Frs2αFlox; CN, Frs2αCN; Solid bars represent 50 μm.