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. 2016 Feb 27;23:30. doi: 10.1186/s12929-016-0229-4

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© Chen et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — BetA promotes TGF-β-induced fibronectin expression. Cells were incubated with 1.25 μg/mL and 2.5 μg/mL of BetA, and then treated with 100 pM TGF-β for 48 h. Whole-cell extracts were prepared and analyzed by Western blot analysis using an antibody against fibronectin (top). The quantitative data from three analyses is shown as mean ± SD (bottom). **The group of BetA/TGF-β co-treatment significantly higher than that in cells treated with TGF-β only (P < 0.01)