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. 2016 Mar;26(3):365–375. doi: 10.1101/gr.196857.115

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© 2016 Peace et al.; Published by Cold Spring Harbor Laboratory Press

This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first six months after the full-issue publication date (see http://genome.cshlp.org/site/misc/terms.xhtml). After six months, it is available under a Creative Commons License (Attribution-NonCommercial 4.0 International), as described at http://creativecommons.org/licenses/by-nc/4.0/.

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Figure 5. — Fkh1-OE increases Fkh1 occupancy at stimulated origins. Strains JPy105 (WT, Fkh1-Myc) and JPy106 (Fkh1-Myc-OE) arrested and induced (but not released into S phase) as described in Figure 2A were subjected to ChIP-chip analysis of Fkh1. (A) ChIP signal along Chromosome XI is plotted with origins indicated above. (B) The average ChIP signal is plotted for 5-kb regions aligned on origins of the indicated groups. (C) Box-and-whisker plots of the distributions of ChIP signals for individual origins within the indicated origin groups.