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. 2016 Mar 3;118(5):856–866. doi: 10.1161/CIRCRESAHA.115.307918

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© 2015 The Authors.

Circulation Research is published on behalf of the American Heart Association, Inc., by Wolters Kluwer. This is an open access article under the terms of the Creative Commons Attribution Non-Commercial-NoDervis License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited, the use is noncommercial, and no modifications or adaptations are made.

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Figure 7. — Proposed mechanism of TERT in regulating balance of endothelial mitochondrial ROS and NO to maintain FMD in human microvessels. AGS 499 indicates telomeres activator; ATP, adenosine triphosphate; CAD, coronary artery disease; FMD, flow-mediated dilation; H₂O₂, hydrogen peroxide; mt, mitochondrial; nuc, nuclear; NAD+, nicotinamide adenine dinucleotide; NO, nitric oxide; O_2–·, superoxide; ROS, reactive oxygen species; and TERT, catalytic subunit of human telomerase complex.