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. Author manuscript; available in PMC: 2016 Mar 1.
Published in final edited form as: Pharmacol Res. 2015 Dec 30;104:115–123. doi: 10.1016/j.phrs.2015.12.032

Fig. 6.

Fig. 6

Role of β2-AR signaling in higenamine-mediated attenuation of caspase-3 cleavage and AKT inactivation induced by ex vivo I/R in mouse hearts. (A) Representative Western blots showing the level of cleaved-caspase 3 (C-caspase-3), phosphorylated AKT at S473 (p-AKT) in sham and I/R hearts treated with higenamine (100 μM) and/or β2-AR antagonist ICI118551 (0.5 μM). (B and C) Quantification of A, *P<0.05 vs. I/R group, #P<0.05 vs. I/R + Higenamine group.