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. 2016 Feb 22;8(2):53. doi: 10.3390/toxins8020053

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© 2016 by the authors; licensee MDPI, Basel, Switzerland.

This article is an open access article distributed under the terms and conditions of the Creative Commons by Attribution (CC-BY) license (http://creativecommons.org/licenses/by/4.0/).

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HA delayed the decay rate of the evoked EJPs at the Drosophila neuromuscular junction. (A) representative EJPs traces in control and in the presence of HA (2.5 × 10⁻⁶ M). (B) mean amplitude of evoked EJPs before and after 3 min of HA application. Evoked EJPs were elicited by stimulating the segmental nerve at 0.1 Hz with the amplitude of the pulse of 5V. The values reported are the mean ± SD. (C) mean amplitude of evoked EJPs at various time points (3, 10, 20, 40, and 60 min) in control and in the presence of HA were normalized to the initial amplitude of evoked EJPs, i.e., n = 7 for the control and n = 14 for HA treated. Statistical significance was determined by Student’s t-test, and significant values were set at * p < 0.05 and ** p < 0.01.

HA delayed the decay rate of the evoked EJPs at the Drosophila neuromuscular junction. (A) representative EJPs traces in control and in the presence of HA (2.5 × 10⁻⁶ M). (B) mean amplitude of evoked EJPs before and after 3 min of HA application. Evoked EJPs were elicited by stimulating the segmental nerve at 0.1 Hz with the amplitude of the pulse of 5V. The values reported are the mean ± SD. (C) mean amplitude of evoked EJPs at various time points (3, 10, 20, 40, and 60 min) in control and in the presence of HA were normalized to the initial amplitude of evoked EJPs, i.e., n = 7 for the control and n = 14 for HA treated. Statistical significance was determined by Student’s t-test, and significant values were set at * p < 0.05 and ** p < 0.01.