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. 2016 Feb 27;8(1):1759091416630220. doi: 10.1177/1759091416630220

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© The Author(s) 2016

This article is distributed under the terms of the Creative Commons Attribution 3.0 License (http://www.creativecommons.org/licenses/by/3.0/) which permits any use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access page (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 5. — Astrocyte-specific deletion of ephrin-B1 triggers accelerated recovery of vGlut1-positive excitatory presynaptic sites in SR area of CA1 hippocampus at 7 dpi. (a) Confocal images show increased ephrin-B1 immunoreactivity (green) in WT (left panel), but not ephrin-B1 KO astrocytes (right panel) in the SR at 3 dpi. Note that the remaining ephrin-B1-positive immunoreactivity (green) in ephrin-B1 KO (right panel) represent ephrin-B1 expression in dendrites of CA1 neurons. (b, c) Graphs show integrated intensity of ephrin-B1 immunoreactivity per GFAP-positive astrocyte (b) and vGlut1-positive puncta per 100 µm² area (mean ± SEM; n = 3–4 mice, two-way ANOVA, followed by Bonferroni post hoc analysis *p < .05, **p < .01, ***p < .001).