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. 2016 Feb 27;8(1):1759091416630220. doi: 10.1177/1759091416630220

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© The Author(s) 2016

This article is distributed under the terms of the Creative Commons Attribution 3.0 License (http://www.creativecommons.org/licenses/by/3.0/) which permits any use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access page (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 8. — Visual depiction of hypothesis. Increased ephrin-B1 immunoreactivity in astrocytes coincide with a reduction in vGlut1-positive glutamatergic innervation of CA1 hippocampal neurons in SR at 3 dpi (middle panel), suggesting that astrocytic ephrin-B1 may regulate synapse reorganization in the hippocampus following CCI. Astrocyte-specific deletion of ephrin-B1 accelerates recovery of vGlut1-positive glutamatergic innervation of CA1 hippocampal neurons in SR after CCI (right panel).