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. 2016 Mar 1;12:9. doi: 10.1186/s12993-016-0093-0

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© He et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — Representative histopathological finding of TNF-α in the injured lung after brain ischemia. a, b Immunohistochemistry staining of TNF-α in type I (brown signal, bold arrow), II lung epithelial cells (brown signal, thin arrow) and macrophagocytes (brown signal, arrow head) in the Sham group (a) and BI group (b). c, d ISH of TNF-α (red signals) was detected in the typeI (ISH, red signals, bold arrow), II lung epithelial cells (ISH, red signals, thin arrow) and macrophagocytes (ISH, red signals, arrow head) in the Sham group (c) and BI group (d). Scale bars a–d, 25 μm