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. 2016 Mar 3;13:56. doi: 10.1186/s12974-016-0522-x

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© Fang et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Decreasing the phosphorylated levels of tau in APP/PS1 mice after inhibition of the Nogo/NgR pathway. The phosphorylation of tau (a–d) and activation of GSK-3β (e, f) in the brain homogenates was quantified with western blot. a, b AT-8. c, d PHF13. Values were reported as mean ± SD. *p < 0.05; **p < 0.01, when compared with the vehicle group, n = 3–6