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. 2016 Mar 2;16:13. doi: 10.1186/s12902-016-0093-y

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© Zeng et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — The Tie2/Akt/eNOS signaling pathway of circulating EPCs in the three groups. There was no difference in the phosphorylation of tie2 a, Akt b and eNOS c of circulating EPCs between prehypertensive and normotensive premenopausal women without diabetes mellitus. The phosphorylation of tie2 a, Akt b and eNOS c of circulating EPCs in prehypertensive and normotensive premenopausal women without diabetes mellitus exceeded that in prehypertensive premenopausal women with diabetes. The expression of tie2 a, Akt b and eNOS c of circulating EPCs in the three groups exhibited no difference. Data are given as mean ± SD. (*P < 0.05 compared with normotension without diabetes mellitus, ^# P < 0.05 compared with prehypertension without diabetes mellitus, n = 20 for each group)