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. 2016 Mar 4;12(3):e1005835. doi: 10.1371/journal.pgen.1005835

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© 2016 Hou et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 9 — TOPP1-AtI-2 complex can modulate ABA signaling through physical interactions with PYL11 and SnRK2s. Binding of TOPP1 and AtI-2 with SnRK2.6 can suppress the kinase activity of SnRK2.6. Such suppression can be released at least partially by PYL11 in the presence of ABA.