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. 2016 Mar 4;12(3):e1005868. doi: 10.1371/journal.pgen.1005868

Fig 4. Znf3 is required for transposon silencing.

Fig 4

(A) Quantitative real-time PCR showed that deletion of Znf3 or Qip1 resulted in markedly increased abundance of transposon-derived transcripts, similar to mutation of other canonical components of the RNAi pathway. Strains used: H99 and JF289a (wild type), YPH348 and YPH351 (rdp1Δ), YPH738 and YSB299 (ago1Δ), XW205 and MF65 (znf3Δ), and SEC1 and SEC3 (qip1Δ). (B) Heat map of differentially expressed genes in a wild type cross versus a bilateral znf3Δ x znf3Δ cross or a bilateral rdp1Δ x rdp1Δ cross. Strains used: H99 and JF289a (wild type), XW205 and MF65 (znf3Δ), YPH348 and YPH351 (rdp1Δ).