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. 2016 Mar 5;9:18. doi: 10.1186/s13045-016-0242-9

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© Kaifie et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Distribution of the MPN subtypes in the registry (n = 466). Classical MPN, PV, ET, and PMF represent 85 % of all subtypes, followed by post-PV and post-ET myelofibrosis and MPN-U. Documentation of CNL, HES/CEL, SM, and MPN with a PDGFR-alpha, PDGFR-beta, or FGFR1-aberration was infrequent (together 2.4 % of all subtypes)