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. 2016 Feb 8;3(1):ofw025. doi: 10.1093/ofid/ofw025

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© The Author 2016. Published by Oxford University Press on behalf of the Infectious Diseases Society of America.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs licence (http://creativecommons.org/licenses/by-nc-nd/4.0/), which permits non-commercial reproduction and distribution of the work, in any medium, provided the original work is not altered or transformed in any way, and that the work is properly cited. For commercial re-use, please contact journals.permissions@oup.com.

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Figure 4. — Comparison of individual cytokines and low-level viral loads (VL) by clinical group. (A) Individual data points over time are shown for each of the cytokines that showed a significant difference between immunological responder (IR) and immunological nonresponder (INR) groups. (B) Samples from the 2-year postsuppressive combination antiretroviral therapy (cART) initiation were retested using a transcription-mediated amplification (TMA) assay for human immunodeficiency virus (HIV) ribonucleic acid (RNA). Results are expressed as log₁₀ viral load based on comparison of 2 replicate TMA results to a standard curve (see Methods). (C) Samples from the 2-year postsuppressive cART initiation were tested using a less-sensitive HIV antibody assay, with results reported as signal to cutoff (S/Co). Horizontal line signifies median, and error bars represent the interquartile range. Abbreviations: IP-10, interferon gamma-induced protein 10; MIP, macrophage inflammatory protein; sVEGFR, vascular endothelial growth factor.