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. 2015 Dec 16;15(2):256–266. doi: 10.1111/acel.12432

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© 2015 The Authors. Aging Cell published by the Anatomical Society and John Wiley & Sons Ltd.

This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Percentage survival of wild‐type (N2) or eat‐2 mutant worms alone or treated with allantoin (A), trichostatin A (B), rapamycin (C) or LY‐294002 (D). Worms that did not respond to touch stimulation were recorded as dead. Both eat‐2 and treated worms of both genotypes showed both a significantly longer lifespan than N2 controls and no significant variation from each other (Table 3). (A) Allantoin‐treated N2 had a 21.9% increase in lifespan. (B) Trichostatin A‐treated N2 had a 22.1% increase in lifespan. (C) Rapamycin‐treated N2 had an 18.9% increase in lifespan. (D) LY‐294002‐treated N2 had a 21.6% increase in lifespan. Results are shown for combined data from two representative trials (full range of results is shown in Table S3).