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. 2015 Nov 27;139(1):259–275. doi: 10.1093/brain/awv341

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© The Author (2015). Published by Oxford University Press on behalf of the Guarantors of Brain.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License ( http://creativecommons.org/licenses/by/4.0/ ), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Delayed treatment of disabled arm muscles with AAV1 expressing human NTF3 reversed neglect and improved walking after stroke in elderly rats. ( A ) Schematic of the seven pairs of stimuli that were used to evaluate tactile impairment. Each test started at level 3 (boxed). ( B ) A photograph showing the position that the sticky labels were adhered to on each rat’s forelimbs. ( C ) After 1 week, the two groups of stroke-injured rats had similar, large sensory scores ( P = 0.48). Treatment with AAV1-hNT3 caused a progressive recovery from this neglect relative to controls [treatment F (1,30) = 5.2, P = 0.030]. ( D ) One week after stroke, the two groups made a similar number of errors with their affected forelimbs ( t -test P = 0.50). Treatment with AAV1-hNT3 caused a progressive recovery of their affected forelimbs relative to controls [interaction of treatment × time F (7,225) = 2.15, P = 0.040].