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. Author manuscript; available in PMC: 2016 Mar 13.
Published in final edited form as: Virology. 2014 Jan 29;452-453:52–58. doi: 10.1016/j.virol.2013.12.039

Fig. 7.

Fig. 7

Growth of VZV ROka or HSV-2 in wild-type and ataxia telangiectasia mutant (ATM) fibroblasts. VZV ROka (A) or HSV-2 (B) was used to infect wild-type (GM07667) or ATM mutant (GM02530) fibroblasts for various periods of time. VZV infections were performed with cell-associated virus containing 250 PFU added to 106 cells; HSV infections were performed with 30,000 PFU of cell-free virus added to 3 × 105 cells (MOI 0.1). At various days after infection the monolayers were treated with trypsin and infected cells were titrated onto melanoma cells. Error bars show standard error. The experiment in panel A with VZV was repeated with similar results.