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. 2015 Dec 7;4(4):627–642. doi: 10.3390/antibiotics4040627

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© 2015 by the authors; licensee MDPI, Basel, Switzerland.

This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution license (http://creativecommons.org/licenses/by/4.0/).

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(A, B) Parental strain USA300 and pur mutants showed no defects in log phase or stationary phases under non-stressed conditions. Pur mutants had defective persistence in exposure to (C) rifampicin (2 μg/mL, >10× MIC), (D) gentamicin (60 μg/mL, >10× MIC), (E) heat (58 °C), and (F) a low pH of 4.0.

(A, B) Parental strain USA300 and pur mutants showed no defects in log phase or stationary phases under non-stressed conditions. Pur mutants had defective persistence in exposure to (C) rifampicin (2 μg/mL, >10× MIC), (D) gentamicin (60 μg/mL, >10× MIC), (E) heat (58 °C), and (F) a low pH of 4.0.