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. 2016 Mar 14;212(6):633–646. doi: 10.1083/jcb.201508068

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© 2016 Randise-Hinchliff et al.

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.rupress.org/terms). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/).

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Figure 9. — Different regulatory strategies lead to large-scale changes in nuclear organization over different time scales. (A–C) Time course after shifting to –inositol (A), +α-factor (B), or –histidine (C). (top) Peripheral localization of INO1 (A), PRM1 (B), and HIS4 (C). (bottom) Percentage of cells in which the two loci were ≤0.55 µm for INO1 versus INO1 in diploid cells (A), PRM1 versus URA3:3×PRE in haploid MATa cells (B), and HIS4 versus HIS4 in diploid cells. (D) Schematic for three distinct mechanisms of regulation of gene positioning. *, P ≤ 0.05; **, P ≤ 0.001 (Fisher exact test) between SDC and inducing condition.