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. Author manuscript; available in PMC: 2017 Apr 1.
Published in final edited form as: Psychoneuroendocrinology. 2016 Jan 12;66:195–204. doi: 10.1016/j.psyneuen.2016.01.008

Table 2.

Correlation matrix (Pearson’s r) among BMI, BDI scores, and cortisol-mediated monocytic TNF production with and without GR and MR blockers.

BMI BDI-C BDI-S BDI-T Δ %mono TNF (CoMIR)
Cort only Mifepris. Spirono.
BDI-C r .020
p .910

BDI-S r .427* .559**
p .010 .000

BDI-T r .308 .860** .848**
p .072 .000 .000

Δ %mono TNF (CoMIR) Cort only r −.396* −.163 −.461** −.414*
p .018 .348 .005 .013

Mifepris. r −.241 .452* .178 .376# .417#
p .280 .035 .429 .084 .053

Spirono. r −.298 .145 .128 .136 −.301 .256
p .178 .520 .571 .547 .173 .263

Combo r −.587** .331 −.432# −.142 .604** .771** .358
p .007 .154 .057 .550 .005 .000 .121
*

Correlation is significant at the 0.05 level (2-tailed).

**

Correlation is significant at the 0.01 level (2-tailed).

#

Correlation is at the 0.10 level (2-tailed).

Body mass index (BMI); Beck Depression Inventory (BDI: C, cognitive/affective; S, somatic; T, total scores). CoMIR, cortisol-mediated inflammation regulation; Cort only, 1μM cortisol with no blockers; Mifepris, cortisol with mifepristone; Spirono, cortisol with spironolactone; Combo, both antagonists; %mono TNF, % TNF-expressing monocytes.