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. Author manuscript; available in PMC: 2017 Apr 1.
Published in final edited form as: Cell Signal. 2016 Jan 7;28(4):294–306. doi: 10.1016/j.cellsig.2016.01.001

Fig. 10.

Fig. 10

Illustration depicting the hypothesis that a variety of Ca2+-dependent signaling inputs may alter PKA activity levels at the plasma membrane by inducing gravin redistribution and, in turn, impact gravin’s role in regulating β2-adrenergic receptors, synaptic plasticity, cellular migration, and vascular barrier function.