TABLE 3.
ORFb | Gene | Fold change | Molecular function |
---|---|---|---|
YER170Wbf | MUP3 | −17.4 | Adenylate kinase |
YPL187Wa | MATα1 | −15.2 | Pheromone |
YAR070C | −10.5 | Unknown | |
YBR116Cce | −9.1 | Unknown | |
YPL205Cd | −6.2 | Unknown | |
YKL202Wbf | −5.6 | Unknown | |
YPR142Cbf | −5.5 | Unknown | |
YNL078Wa | NIS1 | −4.1 | Unknown |
YAR066Wa | SEN34 | −4.0 | tRNA-intron endonuclease |
YPL189Wd | GUP2 | −3.5 | Glycerol uptake |
YJL169W | −3.3 | Unknown | |
YBR099Cc | −3.1 | Unknown | |
YPL163Cac | SVS1 | −3.0 | Protein required for vanadate resistance |
YPR130C | −3.0 | Unknown | |
YDL177C | −2.8 | Unknown | |
YBL040Ca | ERD2 | −2.6 | HDEL sequence binding |
YOL135C | MED7 | −2.6 | RNA polymerase II transcription mediator |
YOR129C | −2.6 | Unknown | |
YBL016Wa | FUS3 | −2.5 | Mitogen-activated protein kinase |
YOR394Wa | −2.5 | Unknown | |
YPL056C | −2.5 | Unknown | |
YPR141Cbc | KAR3 | −2.5 | Microtubule motor |
YPL058C | PDR12 | −2.4 | Xenobiotic-transporting ATPase |
YOR032Cae | HMS1 | −2.4 | Transcription factor |
YNL172W | APC1 | −2.4 | Protein binding |
YBR013Cb | −2.4 | Unknown | |
YHR005Cac | GPA1 | −2.3 | Heterotrimeric G-protein GTPase |
YML027W | YOX1 | −2.3 | DNA binding |
YIL054Wa | −2.3 | Unknown | |
YIL123Wa | SIM1 | −2.3 | Regulation of cell cycle |
YKR022C | −2.3 | Unknown | |
YIL036W | CST6 | −2.2 | Specific RNA polymerase II transcription factor |
YOR338Wae | −2.2 | Unknown | |
YOR366W | −2.2 | Unknown | |
YOL152W | FRE7 | −2.1 | Ferric-chelate reductase |
YJL158Ca | CIS3 | −2.0 | Structural constituent of cell wall |
The microarray results for the sgf11 deletion were compared to those previously reported for deletion of the SAGA components SPT20, GCN5, and SPT3 (27).
Superscript letters indicate transcript types as follows: a, transcripts regulated in the same direction by spt20 deletion; b, transcripts regulated in the opposite direction by spt20 deletion; c, transcripts regulated in the same direction by gcn5 deletion; d, transcripts regulated in the opposite direction by gcn5 deletion; e, transcripts regulated in the same direction by spt3 deletion; f, transcripts regulated in the opposite direction by spt3 deletion.