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. Author manuscript; available in PMC: 2016 Mar 18.

Published in final edited form as: Aquat Toxicol. 2011 May 17;105(1-2):1–12. doi: 10.1016/j.aquatox.2011.03.013

Fig. 8 — (A) Western blots for doses 0.01 ng – 10 ng 17β-estradiol/g body weight in males and females. Each sample is a composite of 3 individuals for each treatment group. Dose-response of vitellogenin protein induction by 17β–estradiol in reproductively inactive (B) males and (C) females from both Tuckerton and Newark Bay. N = 6 per treatment group. Data are reported as mean ± standard deviation. *Significantly different from the Tuckerton at p ≤ 0.05 among each treatment group. All treatment groups were significantly higher than control groups (no vitellogenin detected).

Fig. 8 — (A) Western blots for doses 0.01 ng – 10 ng 17β-estradiol/g body weight in males and females. Each sample is a composite of 3 individuals for each treatment group. Dose-response of vitellogenin protein induction by 17β–estradiol in reproductively inactive (B) males and (C) females from both Tuckerton and Newark Bay. N = 6 per treatment group. Data are reported as mean ± standard deviation. *Significantly different from the Tuckerton at p ≤ 0.05 among each treatment group. All treatment groups were significantly higher than control groups (no vitellogenin detected).