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. 2016 Mar 24;12(3):e1005872. doi: 10.1371/journal.pgen.1005872

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© 2016 Hong et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 1 — A. Genetic interaction between SMC-5/6 complex and HIM-6 was examined by combining smc-5/6 deletion with different him-6 alleles. Progeny viability in % was determined by counting number of viable eggs/total number of eggs laid. The sample size (n) indicates the number of embryos examined for each genotype. Error bars represent standard deviation of the mean. Asterisks indicate statistically significant reduction in embryonic viability in smc-5(ok2421); him-6(ok412), smc-5(tm2868); him-6(ok412), smc-5(ok2421); him-6(e1423) and smc-6(ok3294); him-6(ok412) double mutants (p < 0.005 by two-tailed Student T-test) when compared with him-6(ok412) and him-6(e1423). B. Distribution of RAD-51 foci in wild type, smc-5(ok2421), him-6(ok412), and smc-5(ok2421); him-6(ok412) animals. Zone definitions: 1 Early mitotic, 2 Late mitotic, 3 Transition, 4 Early pachytene, 5 Middle pachytene, 6 Late pachytene.