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. 2016 Mar 29;10:77. doi: 10.3389/fncel.2016.00077

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Copyright © 2016 Bálint, Liposits and Farkas.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution and reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Bar graph summarizing the percentage changes in the frequency and the amplitude of the mPSCs resulting from selective ER agonists and various antagonists. The E2 and the selective ERβ agonist DPN significantly decreased the frequency of mPSCs. Effect of DPN was eliminated by the pretreatment with CB1 inverse agonist AM251. Effect of E2 could be inhibited by antagonizing selectively the ERβ by PHTPP. The selective ERα agonist PPT and the GPR30 receptor agonist G1 had no significant effect on the frequency of mPSCs. The amplitude of the mPSCs presented no change in any of the treatments. *p < 0.05 compare to the control; **p < 0.05 as compared to the change caused by E2 treatment.