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. 2016 Mar 3;31(4):525–534. doi: 10.3346/jkms.2016.31.4.525

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© 2016 The Korean Academy of Medical Sciences.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 4 — Recordings of action potentials in left atrial myocytes from control and type 2 diabetic rats. (A) Representative recordings of action potentials in left atrial myocytes from control (blue) and diabetic (red) rats. (B) Pooled data for APD at 50% (APD50) and 90% (APD90) repolarization in control (n = 11 cells from 3 rats) and diabetic rats (n = 11 cells from 3 rats) rats. (C) Summary data of resting membrane potential (RMP) in control (n = 11 cells from 3 rats) and diabetic (n = 11 cells from 3 rats) rats. (D) Summary data of action potential amplitude (APA) in control (n = 11 cells from 3 rats) and diabetic (n = 11 cells from 3 rats) rats. *P < 0.001 vs. control.