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. Author manuscript; available in PMC: 2016 May 1.
Published in final edited form as: Dev Biol. 2016 Feb 3;412(1):99–113. doi: 10.1016/j.ydbio.2016.01.036

Table 1. Different mechanisms of inducing DNA hypomethyltion cause distinct phenotypes in early zebrafish embryos.

Embryos were injected with the indicated morpholino (MO) or RNA immediately after fertilziation or were treated with 5-aza before the 16 cell stage. Methylation levels were determined by slot blot analysis with anti-5MeC. Dnmt1 protein was determined by Western blot. Not done (ND).

Genetic Approach Change in DNA
Methylation
Embryonic
Phenotype
Dnmt1
Protein

uhrf1 Knockdown (MO) 60% Residual DNA
Methylation
High Stage
Arrest
Increased
dnmt1 Knockdown (MO) 52% Residual DNA
Methylation
None Decreased
5-Azacytidine Treatment 57% Residual DNA
Methylation
Sphere Stage
Arrest
Inhibited
WT-UHRF1 Over-
expression (RNA)
61% Residual DNA
Methylation
Asymmetric
Epiboly
Slightly
Increased
UHRF1S661A Over-
expression (RNA)
43% Residual DNA
Methylation
Asymmetric
Epiboly
Increased
WT-dnmt1 Over-
expression (RNA)
32% Residual DNA
Methylaton
Asymmetric
Epiboly
ND
dnmt1C1109S Over-
expression (RNA)
ND Asymmetric
Epiboly
ND