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. 2015 Jul 2;6(7):e95. doi: 10.1038/ctg.2015.23

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Copyright © 2015 American College of Gastroenterology

Clinical and Translational Gastroenterology is an open-access journal published by Nature Publishing Group. This work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License. The images or other third party material in this article are included in the article's Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by-nc-nd/4.0/

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Levels of LIGHT (homologous to lymphotoxin, exhibits inducible expression, and competes with HSV glycoprotein D for herpes virus entry mediator (HVEM), a receptor expressed by T lymphocytes), HVEM, and lymphotoxin β receptor (LTβR) in liver biopsies from patients and controls. mRNA levels of (a) LIGHT and its corresponding receptors, (b) HVEM and (c) LTβR, in liver biopsies from control subjects (n=7), patients with simple steatosis (n=8), and patients with nonalcoholic steatohepatitis (NASH) (n=6). mRNA levels were quantified by real-time reverse transcription-PCR (RT-PCR), in relation to the control gene GAPDH (glyceraldehyde 3-phosphate dehydrogenase). Data are given as mean±s.e.m. *P<0.05 vs. control subjects.