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. 2016 Mar 20;4(5):e12625. doi: 10.14814/phy2.12625

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© 2016 The Authors. Physiological Reports published by Wiley Periodicals, Inc. on behalf of the American Physiological Society and The Physiological Society.

This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Combined pharmacological AMPK and PPARδ activation increases available substrates and muscle glycogen sparing in trained mice. Blood, gastrocnemius and liver were collected at rest and at the point of exhaustion. (A) Serum glucose, (B) serum TG, (C) serum NEFA, (D) muscle glycogen, (E) liver glycogen, (F) total liver glycogen, (G) muscle NEFA and (H) liver NEFA were measured. Data are expressed as means ± SEM (n = 8–9, at rest; 7–8, at exhaustion). Inter‐group significant differences were analyzed by one‐way ANOVA followed by Newman‐Keuls multiple comparison test while intra‐group differences were analyzed by unpaired Student's t‐test. Asterisk (*), plus sign (+), slash (/) and dagger (†) represent significant difference relative to SED, V, G and A, respectively. Single symbols, P < 0.05; double symbols, P < 0.05 while triple symbols, P < 0.001.