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. 2016 Mar 22;37(5):1199–1208. doi: 10.3892/ijmm.2016.2535

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Copyright: © Jia et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Chronic intermittent hypoxia (CIH) improves cardiac function recovery after ischemia/reperfusion (I/R) in isolated rat hearts. After 20 min of stabilization with Krebs-Henseleit (K–H) perfusate, the hearts were subjected to 30 min of global ischemia and 1 h of reperfusion. (A) Left ventricular developed pressure (LVDP) before ischemia. (B) LVDP recovery (percentage of pre-ischemic LVDP). (C) Representative sections for TTC staining (left) and the average infarct size vs. total area at risk (right). (D) CIH decreased I/R-induced necrosis, as shown by lactate dehydrogenase (LDH) release in the effluent after 60 min of reperfusion. Data are represented as the means ± SEM (n=6 rats/group). ^*P<0.05 and ^**P<0.01 vs. normoxia group.