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Tonometric IOP measurements from WT (A) and KO (B) eyes show no effect of genetic ablation or intravitreal injection of the agonist (GSK101; 75 nM) or antagonist (HC-06; 10 μM) on IOP levels at 1 d (1d PI) or 3 d (3d PI) postinjection. IOP in WT eyes was not significantly different from the IOP in KO eyes, suggesting that TRPV4 is not required for the maintenance of steady-state IOP in healthy mouse eyes.
