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. 2014 Oct 3;24(4):1177–1184. doi: 10.1093/hmg/ddu512

Table 3.

Result of previously reported SNPs

SNP Chr. Chr. location Gene Relative Pos. Case
Control
Ptrenda ORb RAFb Alleleb
11 12 22 11 12 22 Case Control 1 2
rs17674580 18 43309911 SLC14A1 0 5 86 439 28 579 4619 1.8 × 10−4 1.54 0.091 0.061 A G
rs1014971 22 39332623 APOBEC3A −20904 117 256 157 920 2545 1761 0.0074 1.19 0.462 0.420 T C
rs2294008 8 143761931 PSCA 0 241 228 61 2079 2416 730 0.0092 1.20 0.670 0.629 T C
rs9642880 8 128718068 MYC −30247 63 226 241 491 2165 2569 0.039 1.15 0.332 0.301 T G
rs798766 4 1734239 TACC3 0 15 174 341 154 1461 3611 0.056 1.17 0.192 0.169 A G
rs11892031 2 234565283 UGT1A8 0 526 4 0 5143 83 0 0.13 2.11 0.996 0.992 T G
rs1495741 8 18272881 NAT2 14158 55 232 244 559 2394 2628 0.62 1.03 0.322 0.315 A G
rs401681 5 1322087 CLPTM1L 0 229 245 56 2359 2288 579 0.64 0.97 0.663 0.670 G A
rs710521 3 189645933 LEPREL1 28584 298 203 29 2948 1996 282 0.92 0.99 0.754 0.755 T C
rs8102137 19 30296853 CCNE1 −6048 2 89 439 37 834 4355 0.92 0.99 0.912 0.913 G A

We analyzed 539 bladder cancer cases and 5581 controls at GWAS stage. Chr., chromosome; Pos., position in the NCBI Build 36.3.

a

P-values were obtained from Cochrane-Armitage trend test.

b

Allele 1 is a risk allele in the previous GWAS studies. OR was calculated using allele 2 as a reference.