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. 2016 Apr 18;17:15. doi: 10.1186/s12868-016-0251-1

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© Cao et al. 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Effects of GCP II KO on GSH (a), MDA (b), SOD (c), GPx (d). TBI caused significant decreases of GSH level, SOD, GPx activities, and an increase in the MDA level. GCP II KO significantly moderated these changes while not altering the levels in sham controls. Data were represented as mean ± SEM (n = 6 per group); *p < 0.05, versus sham control of the same genotype; ^# p < 0.05, versus injured WT mice