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. 2016 Feb 2;9(2):119–129. doi: 10.1161/CIRCGENETICS.115.001254

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© 2016 The Authors.

Circulation: Cardiovascular Genetics is published on behalf of the American Heart Association, Inc., by Wolters Kluwer. This is an open access article under the terms of the Creative Commons Attribution Non-Commercial-NoDervis License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited, the use is noncommercial, and no modifications or adaptations are made.

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Figure 6. — Mean spectral counts of hemolymph proteins. A, Counts for 2 most abundant proteins found only in the dKlf15^NN mutants hemolymph. B, Proteins showing an increase in the mutant’s hemolymph. C, The most abundant proteins in the hemolymph of wild-type and dKlf15^NN mutants. D, Proteins significantly reduced in the hemolymph of the dKlf15^NN mutants. E, Proteins showing the largest, statistically significant, decrease in the mutant’s hemolymph; n=3 independent hemolymph samples from ≈100 flies of each genotype; *P<0.05, ns indicates no significant difference. SPARC indicates secreted protein acidic and rich in cysteine.